Author Archives: Thomas Braun


The following research paper was published by who is the
foremost authority on understanding the true roll of Vitamin D in maintaining good health.

The World Health Organization’s International Agency for
Research on Cancer recommends avoiding outdoor activities at
midday, wearing clothing to cover the whole body, and daily
use of sunscreen on usually exposed skin [1]. The American
Cancer Society advocates Slip! Slop! Slap! and Wrap! to make
sure skin is covered in clothing or sunscreen and to avoid exposure
to the sun between 10 AM and 4 PM [2]. The U.S. Surgeon
General has issued a Call to Action focused on reducing ultraviolet
(UV) exposure, whether from indoor UV or from the sun
[3]. Though these recommendations, all focused on reduction
of skin cancer, are accompanied by brief acknowledgement of
the importance of vitamin D for health, they persist in urging
avoidance of the sun at the precise times when vitamin D can
be synthesized in the skin—the hours between 10 AM and 3
PM—and suggest that all necessary vitamin D can be obtained
through food and dietary supplements.
These recommendations are understandable from the viewpoint
of preventing the 3.5 million new cases of and 2000
deaths from nonmelanoma skin cancer in the United States
each year [4], but they neglect the fact that we have a long cultural
history of appreciation of the sun and use of UV radiation
for healing purposes. Moreover, they neglect that we have
evolved with physiological adaptations to help protect the skin
from the sun [5] when we are mindful of our exposure and do
not burn. They neglect the fact that increased sun exposure,
based on latitude, has been associated with protection from
several different types of cancer [6–15], type 1 diabetes [16],
multiple sclerosis [17,18], and other diseases [19–23]. They
also neglect the fact that exposure to the sun induces beneficial
physiological changes beyond the production of vitamin D.
Though adherence to the current sun-protective recommendations
would likely result in the reduction of nonmelanoma skin
cancer, that reduction would likely be overshadowed by the
potential reduction in deaths from other cancers and from cardiovascular
disease, which could be achieved by doubling
average blood concentrations of 25-hydroxyvitamin D (25
(OH)D) to 40 ng/mL through a combination of sun exposure
and supplements [24].
The potential harm of sun avoidance and the neglect of its
positive effects on human health led to a seminar, Vitamin D
for Public Health: Integrating Sunshine, Supplements and Measurement
for Optimal Health, presented by GrassrootsHealth at
the University of California San Diego to inform and to help
initiate an action plan to restore a more balanced approach to
solar radiation based on input by the conference speakers.
The healing power of the sun and its use in medical treatment
(heliotherapy) have roots extending back into antiquity.
In the modern era, particularly the first half of the 20th century,
Address correspondence to: Christine B. French, MS, GrassrootsHealth, 315 S. Coast Hwy 101, Encinitas, CA 92024. E-mail:
Journal of the American College of Nutrition, Vol. 0, No. 0, 1–7 (2015)  American College of Nutrition
Published by Taylor & Francis Group, LLC
Downloaded by [Christine French] at 13:23 22 June 2015
heliotherapy was widely used in both Europe and North America,
particularly for the treatment of cutaneous tuberculosis, for
which Niels Finsen garnered the Nobel Prize for Medicine in
1903. Much of this work was done prior to the discovery of
vitamin D and of its synthesis in the skin by UV radiation,
which would have been a principal factor in the recovery from
disease reported a century ago. However, with the discovery of
antibiotics, the era of drug treatment of tuberculosis began in
the 1950s, and heliotherapy fell into disuse and is today virtually
forgotten. A major advantage of antibiotics was the ability
to avoid prolonged hospitalization with its associated expense
and disruption of individual lives. But that was a matter of efficiency,
not efficacy.
Tuberculosis currently afflicts 30% of the world’s population.
The effects—and perhaps the benefits—of heliotherapy in
this disorder, though much less well studied today, extend
beyond the synthesis of vitamin D [25]. We do not know (1)
the relative efficacy of antibiotic treatment and heliotherapy
for various manifestations of tuberculosis and (2) whether vitamin
D, by itself, is sufficient to explain the therapeutic efficacy
of heliotherapy in this disease.
Physiological Responses to Sun Exposure
The best recognized response to sun exposure is elevation
of vitamin D status. Two African tribes, the pastoral Masai and
the hunter–gatherer Hadza, have been shown to have serum 25
(OH)D concentrations averaging 46 ng/mL [26]. Both tribes
live in equatorial East Africa, where humans are thought to
have originated, and have daily sun exposure approximating
that of ancestral humans.
Physiological responses go beyond production of vitamin
D. When the skin is stimulated with UVA radiation, nitric
oxide is released, stimulating vasodilation and lowering of
blood pressure. During active exposure to UVA, diastolic
blood pressure in one study fell by roughly 5 mmHg and
remained lower for 30 minutes after exposure [27]. A reduction
of diastolic blood pressure by 5 mmHg decreases risk for
stroke by 34% and coronary heart disease by 21% [28].
Another physiological response of skin exposure to sunlight
is the thickening of the stratum corneum (the outermost layer
of the epidermis) and increased skin pigmentation through production
of melanin. This paired response actually protects the
skin and deeper tissues from the deeper penetrating and damaging
UVA rays while retaining benefits from UVB exposure
[29]. Though both UVA and UVB exposure result in increased
skin pigmentation, the mechanisms are different, with UVB
being responsible for the up-regulation of melanin synthesis
and thus the protective effects against UV damage to DNA
[30]. The best time for creating this response coincides with
the time of maximal UVB availability (10 AM–3 PM).
Additionally, human skin produces beta-endorphin in
response to UVB exposure [31]; these opioid peptides have the
result of increasing a feeling of well-being, boosting the
immune system, relieving pain, promoting relaxation, wound
healing, and cellular differentiation [31–33]. Light signals
received through the eye regulate production of melatonin and
serotonin for circadian rhythm control and also play a role in
seasonal affective disorder [34].
Impact of Sunlight and/or Vitamin D on Specific
Health Conditions
Studies of the relationship between cancer, sun exposure,
and vitamin D began decades ago with geographic associations
with cancer mortality. In 1941, Apperly reported an association
between latitude and cancer mortality based on sun exposure
although vitamin D was not yet explicitly implicated [6]. In
1980, Garland and Garland reported the association between
latitude and colon cancer using sun exposure as a proxy for
vitamin D status [7]. By 1990 it had been hypothesized that
deficiency of vitamin D was the main cause of breast cancer
In the last decade, analyses of UVB irradiance and cancer
incidence in countries worldwide have shown a regular pattern
of higher rates for countries that are further away from the
equator compared to those near the equator. This pattern has
held true for cancers of the colon [8], breast [9], pancreas [10],
ovary [11], brain [12], bladder [13], kidney [14], and multiple
myeloma [15].
In the United States, mortality rates for 15 types of cancer
for white Americans are highest in the northeast and lowest in
the southwest; these rates are inversely correlated with solar
UVB irradiance [38]. Similar findings have been reported for
Australia, China, Japan, and Spain [39].
In addition to these epidemiological studies, other studies
using serum concentrations of 25(OH)D have found strong
inverse associations for cancer risk and vitamin D status. A
2011 study in 10 European countries reported that individuals
with the lowest concentrations of 25(OH)D (averaging 8 ng/
mL) had almost 3 times the risk of colon cancer as those with
the highest (averaging 50 ng/mL) [40]. For breast cancer,
Lowe et al. found a 50% lower incidence for women who had
25(OH)D concentrations at 48 ng/mL compared to those at
10 ng/mL [41]. Similar findings from Mohr et al. in 2011
showed a 50% reduction in short-term incidence at 45 ng/mL
versus 12 ng/mL [42]. A more recent meta-analysis of 11
case–control studies of breast cancer incidence rate versus 25
(OH)D concentration near time of diagnosis found a 70%
lower incidence rate for 45 ng/mL compared to 5 ng/mL [43].
Though higher latitudes and greater cloud cover predictably
decrease vitamin D status, the inverse association is also true:
lower vitamin D status is a marker for reduced sun exposure.
This distinction is important because it is not possible in the
epidemiological studies cited to distinguish the effects, if any,
Sunlight and Vitamin D
2 VOL. 0, NO. 0
Downloaded by [Christine French] at 13:23 22 June 2015
due to reduced vitamin D status and those due to other actions
possibly produced by sun exposure. The mechanism of heliotherapy
action must be recognized as increased vitamin D synthesis
plus other, inadequately characterized spectral effects.
Two intervention studies have reported vitamin D effects on
cancer. Lappe et al., using a randomized controlled trial (RCT)
design, showed an approximate 70% reduction in all-cancer
risk in postmenopausal women given calcium and vitamin D in
a dose sufficient to raise serum 25(OH)D from 29 to 38 ng/mL,
whereas those given only calcium showed an approximate 40%
reduction in all-cancer risk [44]. In another study, men with
low-risk, biopsy-proven early-stage prostate cancer were given
4000 IU/day of vitamin D3 [45]. Over the year of treatment,
mean circulating concentrations of 25(OH)D rose from 33 to
66 ng/mL. A second biopsy showed that the number of cores
positive for malignancy was reduced for more than half of those
enrolled in the study. Patients from the same practice who did
not receive supplementation showed an increase in positive
cores over the same period of time. There were no adverse
events as a result of vitamin D supplementation in either study.
A proposed model for how vitamin D deficiency is related
to a wide array of cancers is the DINOMIT model of cancer
progression, which outlines a mechanism whereby vitamin D
might restrain cancer development and spread [46].
Type 1 Diabetes
Improved vitamin D status has been associated with a lower
risk of type 1 diabetes. Just as vitamin D status varies by season
and latitude because of availability of sun exposure [47], type 1
diabetes incidence rates peak annually in the winter/spring [48]
and risk varies directly with distance from the equator (just as
with many cancers) [16]. In Finland, with one of the highest
rates of type 1 diabetes, the frequency and dosage of vitamin D
supplementation during the first year of life have been associated
with type 1 diabetes rates. Specifically, there is an almost
90% lower risk of type 1 diabetes by age 31 for individuals regularly
given vitamin D supplements in infancy, versus those
who were not given supplements. Among those who received
supplements, those who received at least 2000 IU/day had an
80% lower risk than those who received less than 2000 IU/day
[49]. From 1965 to 2005, Finland had a dramatic increase in
the incidence of type 1 diabetes; over that same period of time,
the recommended vitamin D intake decreased from 4500 IU/
day to 400 IU/day [50]. Though association does not equal causation,
this phenomenon is difficult to explain in any other way.
A qualitatively similar difference in diabetes risk is reported
from the EURODIAB study [51] in which countries recommending
vitamin D supplementation in infancy had lower type
I diabetes incidence rates by age 15 than those countries not
recommending vitamin D.
In a nested case–control study of U.S. service members,
those with 25(OH)D concentrations <14 ng/mL had 3.5 times the risk of type 1 diabetes compared to those at 40 ng/mL or higher [52]. In a large cohort study, both insulin resistance and fasting insulin levels were inversely associated with serum 25 (OH)D concentration, providing biological plausibility for a contributory role of vitamin D in diabetes [53]. Evidence suggests that improving the vitamin D status of the population could lead to a marked decrease in type 1 diabetes incidence. Pregnancy Only recently have we started to understand the developmental origins of disease and how the perinatal environment affects lifelong health. Worldwide there is profound vitamin D deficiency among pregnant women, yet the role of vitamin D in pregnancy has largely been ignored [54]. Epidemiological data have shown that deficiency during pregnancy causes higher risk of maternal preeclampsia [55–58], gingivitis, and periodontal disease in the mother [59,60] and impaired fetal growth [61,62], impaired dentition [63,64], and increased risk of respiratory syncytial virus infection [65] in the infant. A recent RCT in India showed that women who were given vitamin D supplementation during pregnancy had a 61% lower risk of preterm labor and a 47% lower risk of hypertensive complications compared to participants who were not given supplementation [66]. In another RCT, vitamin D supplementation of 4000 IU/day was shown to be safe and effective in achieving sufficiency (32 ng/mL) for pregnant women and their infants, whereas 400 IU/day was ineffective [67–69]. In fact, women who achieved at least 32 ng/mL had a lower risk of gestational diabetes, preterm birth, preterm labor, preeclampsia, hypertensive disorders of pregnancy, and infection [69]. Overall, there is approximately a 50% reduction in preterm birth when 25(OH)D serum concentrations of 40 ng/mL are attained [70]. With a preterm birth rate in the United States of 11.4% and an associated cost of $26 billion per year [71], achieving an optimal 25(OH)D concentration of 40 ng/mL in pregnant women would greatly reduce this human and financial burden. Vitamin D for Optimal Health In common with many other micronutrients, vitamin D is a necessary but not sufficient factor for key cell-biologic processes. That is, it is an enabler; it must be present for those processes to occur, but it does not, itself, stimulate or cause them. In brief, low vitamin D status does not so much cause disease or dysfunction as it impairs cellular response to both internal and external signals. It is now recognized that essentially every tissue and cell in the body has vitamin D receptors. Furthermore, most cells also have the capability of converting 25(OH) D to its active form, 1,25-dihydroxyvitamin D [1,25(OH)2D], and most of our daily vitamin D consumption occurs in this way [72]. This conversion in the cell allows each tissue to use vitamin D as it is needed. It also follows that, in the absence of Sunlight and Vitamin D JOURNAL OF THE AMERICAN COLLEGE OF NUTRITION 3 Downloaded by [Christine French] at 13:23 22 June 2015 vitamin D, none of our body systems can work at their optimal potential. Thus, it becomes clear that low vitamin D status would inevitably be involved in a wide range of dysfunctions and diseases. To address the issue of how much vitamin D is needed for optimal health, it is necessary first to consider the desired endpoint. If the goal is to avoid a known disease, such as rickets, the amount required will be less than if the goal is to optimize bone health. Moreover, shifting focus to the physiological need for vitamin D offers several criteria for determining need [73], including (1) the intake that minimizes the need for functional adaptation or compensation; (2) the status found in our hominid ancestors; or (3) the amount necessary to support a critical life function. These criteria are elaborated below: 1. One manifestation of adaptive compensation is the elevated parathyroid production that occurs when vitamin D status is low. Vitamin D–mediated intestinal calcium absorption reduces parathyroid activity, and the point at which vitamin D ceases to be a limiting factor in calcium absorption is the point at which parathyroid hormone concentrations are lowest. That occurs when serum concentrations of 25(OH)D are in the range of 48–52 ng/mL [74]. 2. Approximating the vitamin D status of ancestral humans, as noted above, has been done for 2 African tribes. Their average 25(OH)D concentrations (around 46 ng/mL) [26] provide the best available estimate of the level to which human physiology has been fine-tuned by natural selection over the millennia of human evolution. 3. A critical function of vitamin D is passage of the nutrient from mother to child in breast milk. In order to fully support the infant’s need for vitamin D, the mother must have a blood concentration of cholecalciferol (vitamin D3) above 10 ng/mL, which happens only when serum 25(OH)D is above 48 ng/mL [75]. At today’s prevailing vitamin D status values, no D3 is transferred into breast milk. Hence, currently recommended 25(OH)D concentrations are not adequate to support this critical physiological function. In brief, all 3 physiological criteria converge on blood concentrations around 48 ng/mL. Though a 25(OH)D concentration of 20 ng/mL may be sufficient to avoid clinically evident rickets, it is not sufficient to sustain physiological functions and promote optimal health. Vitamin D may come from UV exposure, dietary intake, or supplements. The input from all sources required each day to meet physiological needs and to support optimal health is estimated to be roughly 6000 IU/day [76,77]. However, because of variations in individual ability to produce vitamin D from UV exposure or to absorb it from dietary sources, as well as variations in individual requirements, testing serum concentrations of 25(OH)D remains important. Cost–Benefit Analysis Of the 30 leading causes of death in the United States in 2010, 19 have been linked to low vitamin D status, including various forms of cardiovascular disease, various cancers, diabetes mellitus, Alzheimer’s disease, and falls and fractures in the elderly [78]. If the population of the United States were to increase their vitamin D status to 40 ng/mL, we could expect to see a potential reduction of as much as 336,000 deaths each year (out of 2.1 million deaths attributed to the diseases concerned) [24]. This includes estimated reductions of 180,000 deaths from cardiovascular disease, 20,000 from colorectal cancer, 12,000 from breast cancer, 70,000 from other cancers, and 15,000 from Alzheimer’s disease. In addition to this annual reduction in deaths, the direct costs of care for the associated diseases would be reduced by roughly $130 billion each year. Raising 25(OH)D concentrations appears to be the most efficient and cost-effective way to reduce the burden of disease and increase life expectancy in the United States [24]. Among the reasons vitamin D deficiency is so widespread are the public health messages from the U.S. Surgeon General, the Institute of Medicine, and the World Health Organization, all of whom promote avoidance of sun exposure and covering the skin with clothing or sunscreen when out in the sun. It should be noted that these messages focus mainly on reducing nonmelanoma skin cancer. With a total of 5 million cases of skin cancer treated each year at an annual cost of $8.1 billion, skin cancers result in 13,000 deaths annually. Melanoma, by far the most deadly form of skin cancer, accounts for 70%–75% of those deaths [2,3] and 40% of the costs [3]. Despite public health messages to the contrary, not all skin cancers, particularly melanomas, are directly attributable to moderate sun exposure. Though painful sunburns before the age of 20 seem to be a strong predictor of all types of skin cancer, chronic or lifetime sun exposure is associated with an increased risk of nonmelanoma skin cancers but a decreased risk of malignant melanoma [79]. CONCLUSIONS AND RECOMMENDATIONS The full solar spectrum is essential to optimal health and well-being. Humans are physiologically adapted to produce vitamin D in response to sun exposure, specifically UVB radiation; other regions of the spectrum seem to confer benefit as well. Though some vitamin D comes from our diet (and more recently from supplements), we should not ignore the natural capacity that we possess to produce our own. We are of the opinion that moderate sun exposure (less than the time required to burn) to the arms, shoulders, trunk, and legs should be sought rather than avoided. Once that limited time has been achieved, we agree that covering the skin or seeking shade 4 VOL. 0, NO. 0 Sunlight and Vitamin D Downloaded by [Christine French] at 13:23 22 June 2015 may be appropriate. The benefits of such exposure go beyond production of vitamin D and include other physiological responses to sunlight, still inadequately explored, including release of nitric oxide, production of beta-endorphin, and regulation of circadian rhythms—all important components of lifelong health and well-being. The current policy of sun avoidance is creating probable harm for the general population. Ignorance of the effects of portions of the solar spectrum at wavelengths longer than the ultraviolet is due mainly to lack of suitable measurement tools for cutaneous and systemic responses to those regions. We propose therefore that the U.S. Surgeon General’s office, the World Health Organization, the Institute of Medicine, and other health entities, together or separately, engage in an immediate effort both to define and quantify comprehensively the benefits and harms of sun exposure and to develop the measurement methods needed for their detection and quantification. Following this effort, concrete recommendations for exposure at an individual level that are both safe and beneficial should be created. We also recommend, as an interim strategy, that both sun exposure and vitamin D supplementation be concomitants of drug therapy for tuberculosis so as to garner both whatever benefits may be due to vitamin D and those of heliotherapy that extend beyond its effect on vitamin D status. AUTHORS’ NOTE Videos of all presentations from the seminar upon which this article are based can be accessed at public-health. REFERENCES 1. International Agency for Research on Cancer: “IARC Handbook on Cancer Prevention. Vol. 5: Sunscreens.” Lyon, France: International Agency for Research on Cancer, World Health Organization, 2001. 2. American Cancer Society: Skin cancer facts. Accessed at: http:// cer-facts (last revision 4 April 2015). 3. US Department of Health and Human Services: “The Surgeon General’s Call to Action to Prevent Skin Cancer.” Washington, DC: US Department of Health and Human Services, Office of the Surgeon General, 2014. 4. American Cancer Society: “Skin Cancer: Basal and Squamous Cell.” 2013. (last revision 3 April 2015). 5. Garland CF, Garland FC, Gorham ED: Epidemiologic evidence for different roles of ultraviolet A and B radiation in melanoma mortality rates. Ann Epidemiol 13:395–404, 2003. 6. Apperly FL: The relation of solar radiation to cancer mortality in North America. Cancer Res 1:191–195, 1941. 7. Garland CF, Garland FC: Do sunlight and vitamin D reduce the likelihood of colon cancer? Int J Epidemiol 9:227–231, 1980. 8. Cuomo RE, Mohr SB, Gorham ED, Garland CF: What is the relationship between ultraviolet B and global incidence rates of colorectal cancer? Dermatoendocrinol 5:181–185, 2013. 9. Mohr SB, Garland CF, Gorham ED, Grant WB, Garland FC: Relationship between low ultraviolet B irradiance and higher breast cancer risk in 107 countries. Breast J 14:255–260, 2008. 10. Mohr SB, Garland CF, Gorham ED, Grant WB, Garland FC: Ultraviolet B irradiance and vitamin D status are inversely associated with incidence rates of pancreatic cancer worldwide. Pancreas 39:669–674, 2010. 11. Garland CF, Mohr SB, Gorham ED, Grant WB, Garland FC: Role of ultraviolet B irradiance and vitamin D in prevention of ovarian cancer. Am J Prev Med 31:512–514, 2006. 12. Mohr SB, Gorham ED, Garland CF, Grant WB, Garland FC: Low ultraviolet B and increased risk of brain cancer: an ecological study of 175 countries. Neuroepidemiology 35:281–290, 2010. 13. Mohr SB, Garland CF, Gorham ED, Grant WB, Garland FC: Ultraviolet B irradiance and incidence rates of bladder cancer in 174 countries. Am J Prev Med 38:296–302, 2010. 14. Mohr SB, Gorham ED, Garland CF, Grant WB, Garland FC: Are low ultraviolet B and high animal protein intake associated with risk of renal cancer? Int J Cancer 119:2705–2709, 2006. 15. Mohr SB, Gorham ED, Garland CF, Grant WB, Garland FC, Cuomo RE: Are low ultraviolet B and vitamin D associated with higher incidence of multiple myeloma? J Steroid Biochem Mol Biol 148:245–52, 2015. 16. Mohr SB, Garland CF, Gorham ED, Garland FC: Incidence rates of type 1 diabetes in 51 regions worldwide. Diabetologia 51:1391–1398, 2008. 17. Simpson S Jr, Blizzard L, Otahal P, Van der Mei I, Taylor B: Latitude is significantly associated with the prevalence of multiple sclerosis: a meta-analysis. J Neurol Neurosurg Psychiatry 82:1132–1141, 2011. 18. Grant WB, Garland CF, Holick MF: Comparisons of estimated economic burdens due to insufficient solar ultraviolet radiance and vitamin D and excess solar UV irradiance for the United States. Photochem Phobiol 81:1276–1286, 2005. 19. Rostand SG: Ultraviolet light may contribute to geographic and racial blood pressure differences. Hypertension 30:150– 156, 1997. 20. Krause R, B€uhring M, Hopfenm€uller W, Holick MF, Sharma AM: Ultraviolet B and blood pressure. Lancet 352:709–710, 1998. 21. Kinney DK, Teixeira P, Hsu D, Napoleon SC, Crowley DJ, Miller A, Hyman W, Huang E: Relation of schizophrenia prevalence to latitude, climate, fish consumption, infant mortality, and skin color: a role for prenatal vitamin D deficiency and infections? Schizophr Bull 35:582–595, 2009. 22. Vieira VM, Hart JE, Webster TF, Weinberg J, Puett R, Laden F, Costenbader KH, Karlson EW: Association between residences in U.S. northern latitudes and rheumatoid arthritis: a spatial analysis of the Nurses’ Health Study. Environ Health Perspect 118:957– 961, 2010. 23. Schultz M, Butt AG: Is the north to south gradient in inflammatory bowel disease a global phenomenon. Expert Rev Gastroenterol Hepatol 6:445–447, 2012. JOURNAL OF THE AMERICAN COLLEGE OF NUTRITION 5 Sunlight and Vitamin D Downloaded by [Christine French] at 13:23 22 June 2015 24. Grant WB: An estimate of the global reduction in mortality rates through doubling vitamin D levels. Eur J Clin Nutr 65:1016– 1026, 2011. 25. Wunsch A: Lichtbiologie und lichtpathologie. Erfahrungsheilkunde 55:361–369, 2006. 26. Luxwolda MF, Kuipers RS, Kema IP, Dijck-Brouwer DA, Muskiet FA: Traditionally living populations in East Africa have a mean serum 25-hydroxyvitamin D concentration of 115 nmol/l. Br J Nutr 108:1557–1561, 2012. 27. Liu D, Fernandez BO, Hamilton A, Lang NN, Gallagher JMC, Newby DE, Feelisch M, Weller RB: UVA irradiation of human skin vasodilates arterial vasculature and lowers blood pressure independently of nitric oxide synthase. J Invest Dermatol 134:1839–1846, 2014. 28. MacMahon S, Peto R, Cutler J, Collins R, Sorlie P, Neaton J, Abbott R, Godwin J, Dyer A, Stamler J: Blood pressure, stroke, and coronary heart disease. Part 1, prolonged differences in blood pressure: prospective observational studies corrected for the regression dilution bias. Lancet 335:765–774, 1990. 29. Brenner M, Hearing VJ: The protective role of melanin against UV damage in human skin. Photochem Photobiol 84:539–549, 2008. 30. Miyamura Y, Coelho SG, Schlenz K, Batzer J, Smuda C, Choi W, Brenner M, Passeron T, Zhang G, Kolbe L, Wolber R, Hearing VJ: The deceptive nature of UVA tanning versus the modest protective effects of UVB tanning on human skin. Pigment Cell Melanoma Res 24:136–147, 2011. 31. Slominski AT, Zmijewski MA, Skobowiat C, Zbytek B, Slominski RM, Steketee JD: Sensing the environment: regulation of local and global homeostasis by the skin’s neuroendocrine system. Adv Anat Embryol Cell Biol 212;v, vii, 1–115, 2012. 32. Dalayeun JF, Nores JM, Bergal S: Physiology of beta-endorphins. A close-up view and a review of the literature. Biomed Pharmacother 47:311–320, 1993. 33. Sprouse-Blum AS, Smith G, Sugai D, Parsa FD: Understanding endorphins and their importance in pain management. Hawaii Med J 69:70–71, 2010. 34. Paul KN, Saafir TB, Tosini G. The role of retinal photoreceptors in the regulation of circadian rhythms. Rev Endocr Metab Disord 10:271–278, 2009. 35. Gorham ED, Garland CF and Garland FC: Acid haze air pollution and breast and colon cancer mortality in 20 Canadian cities. Can J Public Health 80:96–100, 1989. 36. Garland FC, Garland CF, Gorham ED, and Young JF: Geographic variation in breast cancer mortality in the United States: a hypothesis involving exposure to solar radiation. Prev Med 19:614–622, 1990. 37. Gorham ED, Garland FC, Garland CF: Sunlight and breast cancer incidence in the USSR. Int J Epidemiol 19:820–824, 1990. 38. Grant WB, Garland CF: The association of solar ultraviolet B (UVB) with reducing risk of cancer: multifactorial ecologic analysis of geographic variation in age-adjusted cancer mortality rates. Anticancer Res 26:2687–2699, 2006. 39. Grant WB: Ecological studies of the UVB–vitamin D–cancer hypothesis; review. Anticancer Res 32:223–236, 2012. 40. Jenab M, Bueno-de-Mesquita HB, Ferrari P, van Duijnhoven FJ, Norat T, Pischon T, Jansen EH, Slimani N, Byrnes G, Rinaldi S, Tjønneland A, Olsen A, Overvad K, Boutron-Ruault MC, Clavel- Chapelon F, Morois S, Kaaks R, Linseisen J, Boeing H, Bergmann MM, Trichopoulou A, Misirli G, Trichopoulos D, Berrino F, Vineis P, Panico S, Palli D, Tumino R, Ros MM, van Gils CH, Peeters PH, Brustad M, Lund E, Tormo MJ, Ardanaz E, Rodrıguez L, Sanchez MJ, Dorronsoro M, Gonzalez CA, Hallmans G, Palmqvist R, Roddam A, Key TJ, Khaw KT, Autier P, Hainaut P, Riboli E: Association between pre-diagnostic circulating vitamin D concentration and risk of colorectal cancer in European populations: a nested case–control study. BMJ 340:b5500, 2011. 41. Lowe LC, Guy M, Mansi JL, Peckitt C, Bliss J, Wilson RG, Colston KW: Plasma 25-hydroxy vitamin D concentrations, vitamin D receptor genotype and breast cancer risk in a UK Caucasian population. Eru J Cancer 41:1164–1169, 2005. 42. Mohr SB, Gorham ED, Alcaraz JE, Kane CJ, Macera CA, Parsons JK, Wingard DL, Garland CF: Serum 25-hydroxyvitamin D and prevention of breast cancer: pooled analysis. Anticancer Res 31:2939–2948, 2011. 43. Grant WB: 25-Hydroxyvitamin D and breast cancer, colorectal cancer, and colorectal adenomas: case–control versus nested case–control studies. Anticancer Res 35:1153–1160, 2015. 43. Lappe JM, Travers-Gustafson D, Davies KM, Recker RR, Heaney RP: Vitamin D and calcium supplementation reduces cancer risk: results of a randomized trial. Am J Clin Nutr 85:1586–1591, 2007. 45. Marshall DT, Savage SJ, Garrett-Mayer E, Keane TE, Hollis BW, Horst RL, Ambrose LH, Kindy MS, Gattoni-Celli S: Vitamin D3 Supplementation at 4000 International Units per day for one year results in a decrease of positive cores at repeat biopsy in subjects with low-risk prostate cancer under active surveillance. J Clin Endocrinol Metab. 97:2315–2324, 2012. 46. Garland CF, Gorham ED, Mohr SB, Garland FC: Vitamin D for cancer prevention: global perspective. Ann Epidemiol 19:468– 483, 2009. 47. Wacker M, Holick MF: Sunlight and Vitamin D: A global perspective for health. Dermatoendocrinol 5:51–108, 2013. 48. Gorham ED, Barrett-Connor E, Highfill-McRoy RM, Mohr SB, Garland CF, Garland FC, Ricordi C: Incidence of insulin-requiring diabetes in the US military. Diabetologia 52:2087–2091, 2009. 49. Hypp€onen E, L€a€ar€a E, Reunanen A, J€arvelin MR, Virtanen SM: Intake of vitamin D and risk of type 1 diabetes: a birth-cohort study. Lancet 358:1500–1503, 2001. 50. Mohr SB, Garland FC, Garland CF, Gorham ED, Ricordi C: Is there a role of vitamin D deficiency in type 1 diabetes of children? Am J Prev Med 39:189–190, 2010. 51. EURODIAB ACE Study Group: Variation and trends in incidence of childhood diabetes in Europe. Lancet 355:873–876, 2000. 52. Gorham ED, Garland CF, Burgi AA, Mohr SB, Zeng K, Hofflich H, Kim JJ, Ricordi C: Lower prediagnostic serum 25-hydroxyvitamin D concentration is associated with higher risk of insulinrequiring diabetes: a nested case–control study. Diabetologia 55:3224–3227, 2012. 53. Heaney RP, French CB, Nguyen S, Ferreira M, Baggerly LL, Brunel L, Veugelers P: A novel approach localized the association of vitamin D status with insulin resistance to one region of the 25- hydroxyvitamin D continuum. Adv Nutr 4:303–310, 2013. 54. Dawodu A, Wagner CL: Prevention of vitamin D deficiency in mothers and infants worldwide—a paradigm shift. Paediatr Int Child Health 32:3–13, 2012. 6 VOL. 0, NO. 0 Sunlight and Vitamin D Downloaded by [Christine French] at 13:23 22 June 2015 55. Halhali A, Tovar AR, Torres N, Bourges H, Garabedian M, Larrea F: Preeclampsia is associated with low circulating levels of insulin- like growth factor 1 and 1,25-dihydroxyvitamin D in maternal and umbilical cord compartments. J Clin Endocrinol 85:1828– 1833, 2000. 56. Hypponen E: Vitamin D for the prevention of preeclampsia? A hypothesis. Nutr Rev 63:225–232, 2005. 57. Bodnar LM, Catov JM, Simhan HN, Holick MF, Powers RW, Roberts JM: Maternal vitamin D deficiency increases the risk of preeclampsia. J Clin Endocrinol Metab 92:3517–3522, 2007. 58. Robinson CJ, Alanis MC, Wagner CL, Hollis BW, Johnson DD: Plasma 25-hydroxyvitamin D levels in early-onset severe preeclampsia. Am J Obstet Gynecol 203:366.e1–6, 2010. 59. Dietrich T, Nunn M, Dawson-Hughes B, Bischoff-Ferrari HA: Association between serum concentrations of 25-hydroxyvitamin D and gingival inflammation. Am J Clin Nutr 82:575– 580, 2005. 60. Dietrich T, Joshipura KJ, Dawson-Hughes B, Bischoff-Ferrari HA: Association between serum concentrations of 25-hydroxyvitamin D3 and periodontal disease in the US population. Am J Clin Nutr 80:108–113, 2004. 61. Brooke OG, Brown IR, Bone CD, Carter ND, Cleeve HJ, Maxwell JD, Robinson VP, Winder SM: Vitamin D supplements in pregnant Asian women: effects on calcium status and fetal growth. Br Med J 280:751–754, 1980. 62. Brunvand L, Quigstad E, Urdal P, Haug E: Vitamin D deficiency and fetal growth. Early Hum Dev 45:27–33, 1996. 63. Purvis RJ, Barrie WJ, MacKay GS, Wilkinson EM, Cockburn F, Belton NR: Enamel hypoplasia of the teeth associated with neonatal tetany: a manifestation of maternal vitamin-D deficiency. Lancet 2:811–814, 1973. 64. Reed SG, King LA, Wingate JS, Murali M, Husley T, Ebeling MD, Hollis BW, Wagner CL: Prenatal vitamin D exposure and developmental defects of enamel and/or early childhood caries. PAS abstract 4516.248, 2011. 65. Belderbos ME, Houben ML, Wilbrink B, Lentjes E, Bloemen EM, Limpen JLL, Rovers M, Bont L: Cord blood vitamin D deficiency is associated with respiratory syncytial virus bronchiolitis. Pediatrics 127:e1513–e1520, 2011. 66. Sablok A, Batra A, Thariani K, Batra A, Bharti R, Aggarwal AR, Kabi BC, Chellani H: Supplementation of vitamin D in pregnancy and its correlation with feto-maternal outcome. Clin Endocrinol (Oxf). 2015. doi:10.1111/cen.12751 67. Hollis BW, Johnson D, Hulsey TC, Ebeling M, Wagner CL: Vitamin D supplementation during pregnancy: double-blind, randomized clinical trial of safety and effectiveness. J Bone Miner Res 26:2341–2357, 2011. 68. Wagner CL, McNeil R, Hamilton SA, Winkler J, Rodriguez Cook C, Warner G, Bivens B, Davis DJ, Smith PG, Murphy M, Shary JR, Hollis BW: A randomized trial of vitamin D supplementation in 2 community health center networks in South Carolina. Am J Obstet Gynecol 208:137e1–e13, 2013. 69. Wagner CL, McNeil RB, Johnson DD, Hulsey TC, Ebeling M, Robinson C, Hamilton SA, Hollis BW: Health characteristics and outcomes of two randomized vitamin D supplementation trials during pregnancy: a combined analysis. J Steroid Biochem Mol Biol 136:313–320, 2013. 70. Wagner CL, Baggerly C, McDonnell SL, Baggerly L, Hamilton SA, Winkler J, Warner G, Rodriguez C, Shary J, Smith PG, Hollis BW: Post-hoc comparison of vitamin D status at three timepoints during pregnancy demonstrates lower risk of preterm birth with higher vitamin D closer to delivery. J Steroid Biochem Mol Biol 148:256–60, 2015. 71. March of Dimes: Accessed at: sion/prematurity-campaign.aspx (Prematurity Campaign Overview, last update for overall campaign November 2014). 72. Heaney RP, Armas LAG: Quantifying the vitamin D economy. Nutr Rev 73:51–67, 2015. 73. Heaney RP: The nutrient problem. Nut Rev 70:165–169, 2012. 74. Ginde AA, Wolfe P, Camargo CA Jr, Schwartz RS: Defining vitamin D status by secondary hyperparathyroidism in the US population. J Endocrinol Invest 35:42–48, 2012. 75. Hollis BW, Pittard WB III, Reinhardt TA: Relationships among vitamin D, 25-hydroxyvitaminD, and vitamin D–binding protein concentrations in the plasma and milk of human subjects. J Clin Endocrinol Metab 62:41–44, 1986. 76. Veugelers PJ, Ekwaru JP: A statistical error in the estimation of the recommended dietary allowance for vitamin D. Nutrients 6:4472–4475, 2014. 77. Heaney RP, Garland CF, Baggerly CA, French CB, Gorham ED: Letter to Veugelers, P.J. and Ekwaru, J.P., A Statistical Error in the Estimation of the Recommended Dietary Allowance for Vitamin D. Nutrients 2014, 6, 4472–4475; doi:10.3390/nu6104472. Nutrients 7:1688–1690, 2015. 78. US Burden of Disease Collaborators: The state of US health. 1990–2010: burden of diseases, injuries, and risk factors. JAMA 310:591–608, 2013. 79. Kennedy C, Bajdik CD, Willemze R, De Gruijl FR, Bouwes Bavinck JN, and the Leiden Skin Cancer Study: The influence of painful sunburns and lifetime sun exposure on the risk of actinic keratoses, seborrheic warts, melanocytic nevi, atypical nevi, and skin cancer. J Invest Dermatol 120:1087–1093, 2003. Received March 11, 2015; accepted April 8, 2015. JOURNAL OF THE AMERICAN COLLEGE OF NUTRITION 7 Sunlight and Vitamin D Downloaded by

Is Common Core a Poster child for anti-federal intervention?

Why is it that there is a division among educators over the value of implementing Common Core to evaluate and improve the ability of our children to learn? Many educators believe it is a good program. Is the real issue the balance between state rights and federal rights? Has the Federal government usurped state rights in so many ways that this is push back? Do dedicated educators that want to do their job well see value, but there are those that don’t want federal guidelines to control the future of education in Arizona and other states?

Does the answer to the poor performance of children in the educational system lie elsewhere? Can children that are under nourished and poorly nourished acquire an inability to learn? Would school behavioral problems disappear if the children could focus on the educational task at hand and not think about being hungry or ill from lack of good nourishment? Would our teachers then accomplish what they want in helping the students excel at learning?

It would be beneficial if Governor Ducey and the Department of Education (Arizona) learn about how schools around the nation are turning underachievers into over achievers by having well-nourished attentive children in the class room.

What follows are the words of Dr. Leslie Matthews, the smartest brain surgeon around who implemented the first program of helping malnourished children learn.

The $1 million annually vitamin D/multivitamin nutrition program (2004-20009) that I started at my sister, Principal Bessie Matthews Gardner’s, Ruleville Central Elementary School in Ruleville, Mississippi in 2004 was the nation’s first according to Vitamin Angels president and founder, Howard Schiffer (see first link and video below).93% of the children in this area lived below the poverty level.

I am very glad to see that this program has grown to many cities and countries around the world since 2004. My simple hypothesis was that malnourished/vitamin deficient children have multiple social behavioral problems, poor school attendance, and can’t properly learn or focus.

“Serve First’s commitment to help reach children in the Mississippi Delta has now grown into a campaign in over 50 major cities and 22 countries around the world,” says Schiffer.

After only two years of receiving nutritional support from Univera, Gardner and school officials reported a drastic drop in student absences. While a total of 1,992 absences were recorded in 2005, that number dropped to just 92 in 2007. That’s over a 1,000% decrease in absenteeism! Suspensions and behavioral problems also decreased.

However, the greatest benefit of a parent-driven vitamin nutrition program was that the school went from being on probation (Level 1) to a Level 4 school (second highest academic level)in 1 year (see third link below).

I recently learned that the inaugural class in 2004 who were 4th graders, just graduated from Ruleville Central High School in May 2013. They have continued their academic projectory with many of them scoring in the high 20’s on their ACT Test (college entrance exam, highest score is 36). Most are going to college. Proper nutrition has lasting, long term positive effects.

1). Ruleville Elementary first program in the nation:

2). Youtube Video of Ruleville vitamin nutrition program 2004/2005:

3). Level 1 to Level 4:

It’s time that Arne Duncan in Washington starts connecting the dots and address the basic core issues that prevent our children from learning.

One Small step for McDonalds.


I was born and raised in Des Plaines Illinois and was a frequent visitor to the first McDonalds. It was when McDonalds sold mostly wholesome food. Over the past 50 years, the quality and nutrient base of the food we consume in the United States has deteriorated. In addition, too many chemicals have been added to the foods we consume. I applaud McDonalds decision to only buy human antibiotic*** free chicken. It is long overdue, and shows that McDonalds can be the leader in delivering more wholesome food to their customers and especially the children. Up until a few years ago, it was common for chicken farmers to give the potent antibiotic Cipro to their chickens. I applaud the Union of Concerned Scientists for taking action and having the practice stopped.
I don’t know why the FDA did not stop the practice.

I suggest that McDonalds should consider the following additional changes in food sourcing.
1. Reject all foods that have a GMO origin. They carry toxins not fit for human consumption
2. Use only non-GMO healthy cooking oils.
3. Eliminate all chemical artificial sweeteners. Offer only natural sweeteners.
4. Eliminate corn syrup based products as much as possible. We consume too much sugar/corn syrup in
this country to the tune of 160 pounds per year per capita.
5. Take the rubber plasticizers out of the bread. We don’t need azodicarbonamide in bread.
6. Use only Vitamin D rich eggs which mean no factory farm eggs from chickens raised in artificial light.
7. Offer more caffeine free drinks for consumption by children. Help prevent ADD.
8. Eliminate Barq’s Root beer with caffeine. It was caffeine free till Coke bought them.
9. Label the amount of caffeine in the drinks you sell.
10. Don’t add dextrose, to your French fries which is converted Acrylamide

Your customers have the right to know what they are consuming and the ability to reject those foods that are unhealthy. McDonalds has taken the first small step in becoming socially responsible and helping improve the health of millions of Americans. I encourage McDonalds to show more leadership and help this nation to become healthy again as it was in the 1950’s.

***The chickens are still being raised in artificial light in crowded indoor barns and the antibiotic ionophores is added to chicken feed which is not approved for human consumption, but is required to prevent chicken diseases to develop because of the crowded conditions that the chickens are raised in. They also add an ingredient to the chicken feed which compensates for the lack of vitamin D due to the chickens being raised in artificial light. It prevents the chickens from having weak bones and breaking legs.

The following video by Michael Polin discusses how McDonalds French Fries

Symphony of Life

My perspective of our very existence has been altered recently. I viewed a new documentary called “The Symphony of the Soil” It explained how soil is alive with microbes that work with the plants to provide the nutrients that the plants require. In turn, we consume the plants that carry some of the nutrients we require. We in turn require microbes in our gut to help facilitate the absorption and production of the nutrients that our cells require. In essence, we come full circle and eventually return to the soil. We must be in concert with the earth to survive.

The American Indian respected our mother earth. Unfortunately, our modern American society does not respect the earth and we are forever poisoning our soil with toxins from our meat and crop growing protocols including the false belief that we can do better than nature. We are adding tons and tons of toxins to our environment as waste products such as CO2** as well as adding insecticides, herbicides and now glyphosates to mother earth that is up taken into the food crops that we consume to stay alive. We are destroying the earth microbes with the toxins that are part of our life cycle. We are creating dead earth. We are the recipients of multiple trace toxins that negatively impact our bio systems. We are also the recipient of dead food without the precious nutrients that we need. We have become a chemical society that shows no respect for mother earth.

As an example, Splenda (sucralose) is found in our water everywhere. It is found in our Ocean water. It is now known that it destroys the good bacteria in our gut. We need good bacteria in our gut to deliver the key nutrients we need. Each molecule of Splenda contains 3 Chlorine atoms. The chlorine atom is a master oxidizer. This is one example out of thousands of how we are negatively impacting our health.

We are an expression of mother earth and need to respect it. I fear that the voices of reason and understanding are being drowned out by those that profit from their efforts to violate this earth. The challenge is how can we identifying an action plan that changes the path this society is on to ruination and our eventual demise?

The adult human is estimated to have 37 trillion cells in its body. Each of these 37 trillion cells has the blueprint for life in its DNA which is composed of 30,000 genes which is an array of only 4 amino acids structured uniquely for each gene. Each cell has been programmed to have a function prescribed by the blueprint for life. The process of existing and replicating requires 100’s of nutrients to be available for the living cell to perform its task properly.

When toxins in varying amounts are introduced into the body through our modern lifestyle which has changed dramatically over the last century, these toxins in trace amounts compete with the essential nutrients for the cell’s needs. In other words, the receptor sites on the cell have a nutrient replaced by a toxic molecule that is similar in design to the nutrient that is supposed to be available at the receptor site.
The nutrient that has been studied most extensively by the scientists in the last 10 years is Vitamin D. Each of the 37 trillion cells in our body have a receptor site for Vitamin D. Research indicates that they have identified over 3000 genes that require the active form of Vitamin D called calcitriol to enter the cell and turn on a specific gene to have the body stay at its optimum health. When the human is 100% healthy, all the cells are in harmony with each other and the mind and the body are in sync. The expression of life is at its fullest and the song of life is seen and heard.

When we do not have the optimal amounts of the nutrients required, the orchestra’s harmony becomes less vibrant and not all the orchestra participants have a role to play. Using Vitamin D as an example, when the blood value drops lesser amount of genes are turned on to provide the vibrancy of life that we are capable of. Discords develop.

Science has recognized that if you don’t have healthy bones you are deficient in Vitamin D. There is general agreement, that you need about 30 ng’s to prevent bone discord. What has not been said is that at this level we only have a band of 10 players singing the song of life rather than a full orchestra.

As we increase the blood value of vitamin D, our song of life becomes richer and more vibrant. At 50 to 60 ng’s where we were a half century ago before the lifestyle changes we have made has caused us in general to lose half the blood value of vitamin D that is essential to a healthy life.

Now that we are experiencing minimal values of Vitamin D, the susceptibility to disease states has increased dramatically, because not only is Vitamin D competing for its receptor site on the cell, but all the other key nutrients have toxins competing for the rest of the receptor sites.

What is emerging out of the research is the fact that to have a harmonious body, we need to double our vitamin D blood level from 30 ng’s to 60 ng’s. If we are fighting cancer which is caused by abnormal cells turned on not by the nutrients, but by a toxin or pathogen, we have great discord within our body and we need to double the blood value of Vitamin D again to help bring harmony back. Vitamin D does not do it alone, but is just the base violin which works with all the other instruments (nutrients) in the orchestra to provide the song of life (strong immune system) which each of us wants to enjoy life to the fullest.
Our food system has continued to deliver us less and less nutritious food laced with too many trace toxins that is overwhelming our ability to cope.
To bring the orchestra up to its full potential, we must reject foods delivering the wrong sheet music in our life. Nature (God) created the perfect human being. We are using herbicides, insecticides, antibiotics, growth hormones, cold tar dyes, artificial flavors, artificial sweeteners, and many more toxins to produce the food we consume today. Man is destroying the symphony of life by having the wrong concept of what is food.

My mission is to share with everyone what I have learned about our less than healthy lifestyle in the US.

What the FDA is not telling you about Acetaminophen (#1 Brand Tylenol)

It is my professional opinion that individuals who chronically take daily doses of acetaminophen with or without alcohol risk the depletion of their bodies’ store of an essential amino acid called glutathione.
I maintain my glutathione level by taking Ultra Glutathione 500 mg daily which I have found to be bio available. Read my article called:


The chemical was discovered around 1900. It has no anti-inflammatory properties. It sat on the chemist’s shelf as an oddity until the 1950’s when J&J decided to market it as a competitive product to aspirin. The marketing advantage was that it reduced the risk of stomach ulcers.
Physicians bought into the concept big time. The pain pill was legitimized by the FDA because it was marketed originally only to physicians as an Rx item. Once J&J had the marketing information that stated “Tylenol is the number one recommended pain pill by physicians” the product was converted to a non-prescription status. Fast forward to the 1980’s and an Australian physician determined that the major culprit in stomach ulcers was H. Pyloric. It took 10 years for our medical profession to accept his findings as being legitimate.

Our livers today are being over taxed by a myriad of trace toxins that need to be metabolized by our livers. When overdosing occurs, essential nutrients are depleted. Glutathione is a key one that is destroyed by excess acetaminophen consumption. In addition, it is converted to phenacetin in the body and will crystalize in the kidney and damage the nephrons if the individual is dehydrated. The FDA took phenacetin off the market, but no one at the FDA looked at how acetaminophen is metabolized in the body.

There is a current theory that the FDA is aware of, that consumption of acetaminophen creates a gliotoxin that causes autism in the infant. Paregoric has been replaced by Tylenol baby drops over the last 50 years. Read my “Autism Crisis N2E”  article. Major cultural changes have taken place over the last fifty years or so. Considering the heartache of parents who have an autistic child and the number of people who have required a liver transplant due to acetaminophen poisoning why do we need it except under physician’s supervision?

Also consider the fact that many millions overdosed over the last 50 years. Some of which died for lack of a liver being available or just not seeking medical help in time. A mother whose son had the flu died of self-medicating on multiple acetaminophen products pleaded with the FDA to prevent this from happening in the future. It fell on deaf ears back in the 1990’s.I believe our medical leadership lacks compassion, empathy and real concern for preventing unnecessary pain and suffering due to the over use of OTC and Rx drugs in the US. Acetaminophen is the poster child of bad medicine. When will bad drug marketing stop trumping good medicine?

Contact me if you are interested in obtaining this nutrient.

The Ugly Side to Food Dyes by Lisa McGill

The children’s food industry knows exactly what sells. Children like attractive packaging, bright colors and vivid, sugary fruit drinks with a hue so artificial they don’t look like they’ve ever been near a real fruit. Judging by the worldwide renewal of interest in organic foods, parents are beginning to realize the damaging health impact that pesticides and additives have on their children’s food. Unfortunately on many occasions the children are present for the weekly shopping trip and so influence parental buying decisions. Shop displays are often aimed at children with items that would appeal to them placed on lower shelves within easy reach or near to checkout tills so they can pester their parents on the way out for that extra purchase and children are bombarded with commercials on TV, billboards and in magazines every day. It is how junk sells and it’s a strategy that has worked for decades.

The ADHD Epidemic

At the same time, the rate of attention deficit hyperactivity disorder has grown by 24% in only one decade and demand for the drug, Ritalin, has sky rocketed. Of all the countries in the world, the US consumes 85% of the supply of Ritalin. Through 1991-1999 the prescription rate for Ritalin climbed by an unbelievable 500%. Now, 10% of both high school aged boys and girls in the US are taking medication for ADHD.

What is even more concerning about these startling statistics is that the drug Ritalin is an amphetamine-like drug and it acts on the brain in the same way as an amphetamine or cocaine by stimulating dopamine, a neurotransmitter in the brain responsible for the feeling of pleasure. It is addictive in people who use it heavily and prescriptions can be procured for the purposes of illicit use, which is what led to it being banned in Sweden.

Obsessive Compulsive Disorder in Children

In addition to children with ADHD, There are also up to 500,000 children and teens that have obsessive compulsive disorder (OCD) in the US – a condition where the child thinks obsessively about the same subject again and again or they are fixated on performing the same activity repeatedly. They may also feel anxious as if they are in danger and not be able to rid themselves of the feeling. The sheer number of young people with OCD matches that of young people with diabetes and means that for every average sized elementary school there will be four or five children enrolled who have OCD. Before reaching for the bottle of pills to calm down a child that is bouncing off the ceiling, parents should look in their cupboards at the food they buy because chances are what the child is eating is contributing to or even causing the disruptive behavior.

Food Dyes, Hyperactivity and Bad Behavior

Food dyes are a number one culprit. In a study of almost 300 children, researchers tested the ability of food additives to cause hyperactivity. They gave a group of children two types of fruit drink containing the preservative sodium benzoate and various food colorings including:

Sunset yellow dye

Quinoline yellow dye


Allura red dye

They drank the preservative and dye laced drinks for a week with the three year olds having 300mls a day and the eight to nine year olds having 625mls a day. In a separate week, the children were given a placebo drink that didn’t contain the chemicals and their parents and teachers were asked to record their attention levels on a specially designed computer test.

Both parents and teachers found that when children had the additive drinks – containing enough dye andpreservativesfor two to four 56g bags of sweets –it had a significant negative effect on their behavior. Even when the amount of drink consumed was restricted, the negative behavior still occurred.

Endocrine Disrupting Dyes

Parents would be forgiven for thinking that it’s just sweets and cordial that is the problem. Dyes on any type of food have potential health risks. You might have bought your child a nice piece of fish, thinking that is healthy and will give him his omega 3, but what you didn’t know is, many varieties of fish are actually sprayed with dye so that they retain their pink fleshy look. It makes them more visually appealing to the customer. People don’t want to eat gray fish and it would affect sales – but if they were aware of what goes into their food they might change their mind.

Sometimes a preservative is used to prevent the fish from losing its color after death. This preservative – 4-hexylresocrinol – has been used routinely but has only recently been discovered to be an endocrine disruptor. It actually mimics the action of estrogen in the body. This can have numerous unintended health consequences such as infertility and cancer.

Due to research into the harmful effects of food dyes, the UK government has requested that all food manufacturers remove artificial colorings from their products but the US hasn’t followed suit. There are over 3,000 food additives and dyes added to food in the US and none are safety tested to see if they disrupt hormones. Given the vast number of functions that hormones have in regulating every aspect of our physical body and our emotions and behavior, it seems almost incredible that this vital information is omitted from safety tests. Even more incredible is the fact that health agencies rarely consider the huge array of chemicals ingested by children every day that may be impacting negatively on their physical and emotional development.

Choose Organic for your Child

If you want to avoid hyperactivity in your child, go back to basics. Instead of buying pre-packaged meals, cook your own from fresh, local produce so that you know what is going into your family food. It is more time consuming but it’s worth it. Choose organic so that you can be sure there are no pesticides or herbicides in your food. Organic food manufacturers usually do not add any dyes to their produce. Cut out the excess sugar, candy and fruit cordials from your child’s diet and opt for real fruit juice or filtered water instead. Then sit back and see if your child’s concentration and school work improves.

Achieving State of Good Health


Macro View of Good Health











Thomas A. Braun RPh Founder
N2E Health Education Institute, LLC cell 847 370 9080

N2E+ for Life


N2E+ for Life Video cover

A video presentation that explores the need for health education to reverse the dysfunctional healthcare system in the United States. Learn why the driver of this crisis is the nutritional system as it is currently managed by the Department of Agriculture and the National Institue of Health, as well as the Institute of Medicine and the Food and Drug Administration. Learn that a balanced approach to Nutrition, Nutrients and Exercise plus Mind-Body Harmony is key to a long healthy life. The bottom line is that each individual has to reject nutrient deficient dead foods as well as toxins in our food and drink as well as the environment. Positive change only takes place through an understanding of the unhealthy aspects of the American life style. Come and learn.

This video ($20 value) is free and only shipping and handling charges apply.
Email to learn how to order this important and timely free video.